TRANEXAMIC ACID

Tranexamic acid is an antifibrinolytic that blocks plasminogen activation, reducing bleeding in surgery and trauma. Side effects include nausea, seizures (high doses), thrombosis risk, and visual disturbances. Only GMP materials will be supplied, logistics all according to GDP.

SKU: 1e592b5b912a Category: Tag:

Product Description


Mechanism of Action

TRANEXAMIC ACID demonstrates a multilayer biochemical interaction architecture integrating highbandwidth signalling interference, catalyticdomain perturbation, mitochondrialnetwork recalibration, ionflux redistribution, cytoskeletal restructuring, membranepotential modulation and transcriptionfactor pathway realignment. Its molecular conformation enables docking to catalytic residues, allosteric nodes, transmembrane helices, nucleotidebinding pockets, redoxbuffer centres and polymeric scaffolding complexes, providing systemwide influence across metabolic, electrophysiological, structural and genomic domains.

At the signalling stratum, TRANEXAMIC ACID may reshape phosphorylation landscapes, alter the propagation kinetics of ERK, MAPK, JNK and p38 pathways, modulate PI3KAKT survival gating, adjust Gprotein coupling efficiency, redistribute Ca²⁺ microdomains, influence IP/DAG signalling geometry, and redefine cAMPPKA amplitude. Mitochondrial effects include ETCcomplex rebalancing, ATP/ADP cycle remapping, ROSthreshold displacement, membranepotential polarity modulation and crossorganelle stress signalling.

HighPrecision

  • Kinomescale cascade interference mapping & catalyticpathway reconstruction
  • Highresolution docking, ligandmetastability tracking & conformationalflow modelling
  • UPR/ERstress, mitophagy and organelle crosstalk quantification
  • Multiomics regulatorynetwork reconstruction (RNAseq, metabolomics, phosphoproteomics, proteomics)
  • Cytoskeletal mechanics including actin/tubulin turnover and forcedistribution modelling
  • Cellfate simulations across apoptosis, necroptosis, ferroptosis & parthanatos pathways
  • AIenhanced SAR/QSAR optimisation engines for predictive compound modelling

Toxicodynamics & Cellular Hazard Spectrum

  • Acute ROS surge and antioxidantbuffer saturation
  • Mitochondrial fragmentation or ETCaxis collapse
  • Severe Na⁺/K⁺/Ca²⁺ ionhomeostasis destabilisation
  • Cytoskeletal depolymerisation & structuralintegrity failure
  • Membranetension breakdown & lipidbilayer thinning
  • Hyperactivation of inflammatory master regulators (NF-κB, STAT, IRF families)
  • Activation of multiple programmed-cell-death pathways
  • Epigenetic drift including methylation and acetylation instability

For expert not intended for biological or therapeutic exposure.

Only GMP materials will be supplied, logistics all according to GDP.

Datasheet


Molecular Formula

C8H15NO2

Molecular Weight

157.21 g/mol

CAS Number

701-54-2

Storage Condition

Store in a cool, dry place. Keep container tightly closed. Protect from moisture and light.

Solubility

Solubility depends on solvent and conditions (e.g., pH). Please contact us for solvent-specific guidance.

Purity

Purity information is available upon request (COA).

Synonym

701-54-2; 4-(Aminomethyl)cyclohexanecarboxylic acid; Cyclohexanecarboxylic acid, 4-(aminomethyl)-; 4-(Aminomethyl)-Cyclohexanecarboxylic Acid; DTXSID50904827

IUPAC/Chemical Name

4-(aminomethyl)cyclohexane-1-carboxylic acid

InChl Key

GYDJEQRTZSCIOI-UHFFFAOYSA-N

InChl Code

InChI=1S/C8H15NO2/c9-5-6-1-3-7(4-2-6)8(10)11/h6-7H,1-5,9H2,(H,10,11)

References

https://pubchem.ncbi.nlm.nih.gov/compound/5526;

3D Conformer.

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